CSI MEDBLOGS: FURTHER INVESTIGATION OF CT BRAINS TURNS UP NEW ASTOUNDING EVIDENCE

Terri Did Not Suffer Her Brain Damage Outside The Hospital--It Occurred While She Was Hospitalized

According to a time-line of physicians and therapists reports, posted on The Empire Journal, Terri's injury occurred on 2/25/90:

2/25/1990		Terri's Injury
2/25/1990	X-Ray Report Dr. Hameroff	Images taken of cervical spine, no acute bony pathology - straightening of normal cervical lordosis
2/25/1990	Neuro Consult Dr. DeSousa	Deeply comatose.  No evidence of acute process in CT scan.  Evidence of myoclonic seizures.  Rule out acute myocardiac infarction, seizures as cause.  Neck is somewhat stiff as is all of the muscles of the body.  No jugular venous distension.  WBC 26,300, drug screen negative
2/25/1990	CT Scan Report Dr. Hameroff	Normal
2/26/1990	EEG Report Dr.  DeSousa	Abnormal EEG indicative of generalized suppression and slowing
2/27/1990	DeSousa Report	Brainstem Auditory Evoked Response shows no significant disruption of the brain stem acoustic pathway - study within normal limits
2/27/1990	CT Scan Report Dr. Greenberg	Normal CT Scan
2/28/1990	EEG Report Dr. DeSousa	Generalized diffuse slowing.  Some fast frequency rhythms which were not obvious during previous recording.  May have been slight improvement over last EEG.
3/19/1990	EEG Report Dr. DeSousa	Markedly abnormal EEG, no significant improvement from previous records
3/30/1990	CT Scan Report Dr. Abramson	CT Scan shows noncommunicating hydrocephalus, changes occurred since 2/27 exam

If you look carefully at this time line you will see that Terri had "NORMAL" CT's of the brain on 2/25 and 2/27.

STOP THE PRESSES

There is categorically and absolutely NO WAY Terri could have suffered MASSIVE ANOXIC INJURY TO THE BRAIN ON 2/25 AND HAVE NORMAL CT SCAN ON 2/27

NO WAY

Brain edema begins to occur about 20 minutes after infarction and by 24 hours her brain (if she HAD suffered a massive anoxic event) would have been MASSIVELY SWOLLEN -- something that could NEVER be missed and NEVER called normal.

NEVER.

If Terri's brain CT was NORMAL 2 days after she entered the hospital than there is NO POSSIBLE WAY she suffered a massive infarction or global ischemia on 2/25.

THIS IS ALL WRONG

Now look at 3/30. Suddenly she develops NONCOMMUNICATING HYDROCEPHALUS.

WHAT?

Did anyone ask HOW? How did she develop noncommunicating hydrocephalus suddenly on 3/30/90 with 2 normal CT scans on 2/235 and 2/27??

CONCLUSIONS?

1. IF this is an accurate report (normal CT brain on 2/27 -- injury on 2/25) then  TERRI DID NOT suffer an event of massive ischemia on 2/25. tHERE IS NO RADIOLOGIST OR NEUROLOGIST OR NEUROSURGEON IN THE WORLD THAT WOULD DISPUTE THIS. it is impossible. tHE ct ON 2/27 WOULD HAVE BEEN grossly ABNORMAL.

2. IF TERRI DID NOT SUFFER ANOXIC DAMAGE ON 2/25 THEN THE REASON FOR HER BRAIN ATROPHY WAS CAUSED BY SOMETHING THAT OCCURRED after 2/25 namely in the hospital during February or march of 1990.

3. How does one develop NONCOMMUNICATING HYDROCEPHALUS in ONE MONTH? By a blood clot obstructing the CSF outflow from the brain at the Foramen of Magendie.

4. How does one get #3.

BY BEING HIT ON THE HEAD AND SUFFERING INTRACRANIAL HEMORRHAGE.

So Terri WAS HIT ON THE HEAD OR DROPPED ON HER HEAD DURING LATER FEBRUARY OR EARLY MARCH WHILE IN THAT HOSPITAL.

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