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Listed below are links to weblogs that reference CSI MEDBLOGS: A THEORY: DID THE FRENCH KILL YASSER ARAFAT?:

» Medblogs Grand Rounds 7, Here! from GruntDoc
Welcome to Medblogs Grand Rounds, a weekly rotating compendium of posts by the medically minded. Here you'll find writings from many different perspectives about medicine, patients, and thoughts about the medical parts of comic books (really). Thus far... [Read More]

» Arafat...um... from The MUSC Tiger
Okay so apparently Arafat is dead...and yet not dead. A Small Victory has a theory on how that's possible. Here is a hint. Digger has his own theory behind Arafat's death and a roundup of other coverage. Jeff Quinton and Rusty Shackleford have ... [Read More]

» What Killed Yasser Arafat? from OpinionBug.com
CodeBlueBlog has an interesting theory about what happened to Yasser Arafat, go check it out here » Word is, when Arafat is 'finally' declared dead he will be buried in Ramallah—Not Jerusalem as some had desired; Read the story here »... [Read More]

» CodeBlueBlog: Did the French Kill Arafat? from Amy Ridenour's National Center Blog
CodeBlueBlog has more on the Arafat diagnosis mystery. [Read More]

» One Can Only Hope... from Cranial Cavity
... someone did. Via Code Blue Blog: DID THE FRENCH KILL YASSER ARAFAT? Can you say, who gives a rats ass in a ferris wheel, as long as the deed is done? [Read More]

» Yasser Arafat Dies At 75 from antifaust
After a long stuggle, Yasser Arafat, President of the Palestinian Authority, dies at 75. The medical condition that took his life is still shrouded in mystery, but CodeBlueBlog has an interesting theory.... [Read More]

» Medblogs Grand Rounds 7, Here! from GruntDoc
Welcome to Medblogs Grand Rounds, a weekly rotating compendium of posts by the medically minded. Here you'll find writings from many different perspectives about medicine, patients, and thoughts about the medical parts of comic books (really). Thus far... [Read More]

» Medblogs Grand Rounds 7, Here! from GruntDoc
Welcome to Medblogs Grand Rounds, a weekly rotating compendium of posts by the medically minded. Here you'll find writings from many different perspectives about medicine, patients, and thoughts about the medical parts of comic books (really). Thus far... [Read More]

Comments

Jeremy

Wow...I think I understood most of that. I guess my money is being spent well here in Charleston. lol

Nice theory by the way.

caltechgirl

Interesting theory, and a screw-up and subsequent cover up wouldn't surprise me. What do you think about the speculation in some quarters that Arafat has AIDS?

DemonPoet

Eureka!

One of the better theories I have seen.

polyphon

I think that Arafat could very well be an alcoholic and has now develooped end-stage alcoholic cirrhosis after many years of chronic active cirrhosis.. This could explain also the sudden jaundice, the thrombocytopenia, the cerebral hemorrhage. Also his viral illness could have been the trigger tipping him over to end-stage liver diease - surely you can't rule out hepatitis A, B, or C either.

Remember when he was holed up in his building several months ago with the Israeli's threatening to take him out? That could have been a time when he might have been drinking heavily to calm himself.

I think if the diagnosis was a type of cancer like metastatic pancreatic, hepatic CA, or cholangioCA, this would have been more easily found - either with serum markers and/or abdominal CTs. If the diagnosis were carcinoma, there would be much less secrecy about it also. The big mystery about his diagnosis makes the diagnosis either of two things: either Arafat was poisoned with a hepatotoxin (and the pro-Israelis are trying to avoid making him a martyr), or Arafat is an alcoholic (and the pro-Arafat faction is trying to hush this story up).

CBBMD

"End-stage" cirrhosis is a disease that is easy for most physicians to recognize and it evolves over a relatively long period of time. The patients have protuberant abdomens and wasted extremities and their lab work is pretty uniformly diagnostic. So, my point is, there wouldn't be any mystery in the diagnosis and I don't think the French could hide it.

As I said above all they had to do was to say that he GOT the cirhosis from a parasite (schistosomiasis, which is very common in the Middle East and often causes cirrhosis)and no one would ask any more questions. Cirrhosis is a final-common-pathway result of many different pathological processes, so, no one would need to know about any alcohol use.

Besides, the CT would show a small shrunken liver, and, if he has portal hypertension (blood back up from deranged liver cells) one can see varices in the splenic hilum, retroperitoneum and many other anatomic areas...so...again, that would eventually come out and they would be foolish to say the CT is negative, then have to defend that later.

Whereas hepatitis can look NORMAL on a CT (which is what they said in a press release -- all CT's normal.

I don't think cirrhosis explains cerebral hemorrhage, and I'm not buying a SPONTANEOUS cerebral hemorrhage, but I'll POST about that later today.

Maybe we should run a contest an this to see who gets closest!

Thanks for posts

CodeBlueBlogMD

polyphon

I think the French are hiding the diagnosis for political reasons. I think the diagnosis is no mystery to them. There are few reasons to hide the diagnosis of cancer, but if it were something that could be construed as exposure to a hepatotoxin (like acetaminophen or mushroom poisoning), or if it were alcoholism there would be plenty of political reasons to hide it. I think none of us got a chance to see how bloated Arafat's belly was but I sure did see his tremors, his jaundice, his wasting away appearance shortly before his "illness." My bet is still on him being an alcoholic.

David Caskey

If you look up arsenic poisoning it might explain many of Arafat's problems.

Anne Haight

It would be soooo karmic if Islamist terrorists end up seeing France as responsible for killing Arafat when he had an otherwise treatable illness. I wonder when France will start blowing up?

imain

i think the french did there best with yasser arafat ,arafat was a great man and i am sure that the doors of heaven would be open wide waiting for hi
"ina lillahi wa ina illaihy ragioun"

imain

i think the french did there best with yasser arafat ,arafat was a great man and i am sure that the doors of heaven would be open wide waiting for hi
"ina lillahi wa ina illaihy ragioun"

Robert Stewart

get a clue here he died of radiation sickness whether he was killed by a neutron beam or x ray laser bu us or the Israeli's or exposed due to some dirty bomb or nuke project of the terrorists is not clear but he almost certainly died of radiation poisoning
CDC Radiation Emergencies | Acute Radiation Syndrome: A Fact Sheet for Physicians


The Merck Manual of Diagnosis and Therapy
Section 20. Disorders Due To Physical Agents
Chapter 278. Radiation Reactions And Injuries
Topics
[General]

[General]
Radiation reactions and injuries: Damage--acute, delayed, or
chronic--to body tissues produced by ionizing radiation.
Ionizing radiation (eg, x-rays, neutrons, protons, or particles,
-rays) damages tissue directly or by secondary reactions. High doses
of radiation can produce observable somatic effects within days.
Many years later, DNA changes due to smaller doses may lead to
chronic disease in exposed persons or to a genetic defect in their
offspring. Relationships between the degree of damage and the
healing or death of a cell are complex.
Harmful sources of ionizing radiation include high-energy x-rays
used for diagnosis and therapy, radium and other naturally occurring
radioactive materials (eg, radon), nuclear reactors, cyclotrons,
linear accelerators, alternating gradient synchrotrons, sealed
cobalt and cesium sources for cancer therapy, and numerous other
artificially produced radioactive materials used in medicine and
industry.
Large amounts of radiation have accidentally escaped from reactors
several times--eg, the well-publicized accidents at Three Mile
Island in Pennsylvania in 1979 and at Chernobyl in the Ukraine in
1986. The latter resulted in > 30 deaths and many radiation
injuries; significant radiation was detected in most of Eastern
Europe and in parts of Western Europe, Asia, and the USA.
Commonly used units of measurement are the roentgen, gray, and
sievert. The roentgen (R) is the amount of x or ionizing radiation
in air. The gray (Gy) is the amount of energy absorbed by a tissue
or substance and applies to all types of radiation. The R and the
centigray (cGy) are essentially equivalent. The sievert (Sv) equals
the Gy adjusted by a quality factor to account for the biologic
effect. It is used because different types of radiation have
different biologic effects for a given amount of energy; eg,
neutrons have a greater effect. For x and radiation, the Sv equals
the Gy. The Gy and Sv have replaced the rad and rem (Gy = 100 rad;
Sv = 100 rem) in current nomenclature. Radiation is often
characterized in the lay press as low level (0.2 to 0.3 Gy) or high
level (> 0.3 Gy). Medical doses are usually 6 Gy given in a very short time is almost certainly fatal.
In contrast, tens of Gy can be tolerated when delivered over a long
period to a small area of tissue (eg, for cancer therapy).
Distribution of the dose within the body is also important.
Generally, the more rapid the turnover of the cell, the greater its
sensitivity to radiation. Most sensitive are lymphoid cells,
followed by (in descending order) gonads, proliferating bone marrow
cells, bowel epithelial cells, epidermis, hepatic cells, epithelium
of lung alveoli and biliary passages, kidney epithelial cells,
endothelial cells (pleura and peritoneum), nerve cells, bone cells,
and muscle and connective tissue cells. During radiation therapy,
vulnerable areas (eg, bowel, bone marrow) are shielded so that high
whole-body doses, which would otherwise be fatal, can be used.
Pathophysiology
At sufficiently high doses, cell necrosis occurs. High but sublethal
doses may interfere with cell proliferation by decreasing the rate
of mitosis, by slowing DNA synthesis, or by causing cells to become
polypoid. In tissues that normally undergo continual renewal (eg,
bowel epithelium, bone marrow, gonads), radiation produces
dose-dependent progressive hypoplasia, atrophy, and eventually
fibrosis. Some cells, injured but still capable of mitosis, may pass
through one or two generative cycles, producing abnormal progeny
(eg, giant metamyelocytes, hypersegmented neutrophils) before dying.
The somatic and genetic effects of doses 30 Gy), is always fatal. It consists of three
phases: a prodromal period of nausea and vomiting; listlessness and
drowsiness, ranging from apathy to prostration (possibly due to
nonbacterial inflammatory foci in the brain or to radiation-induced
toxic products); and tremors, convulsions, ataxia, and death within
a few hours to a few days.
The GI syndrome is produced by whole-body doses of >= 4 Gy. It is
characterized by intractable nausea, vomiting, and diarrhea that
lead to severe dehydration, diminished plasma volume, and vascular
collapse. The GI syndrome results from tissue necrosis and is
perpetuated by progressive atrophy of GI mucosa. Bacteremia due to
necrotic bowel also occurs. Ultimately, the intestinal villi are
denuded, with massive loss of plasma into the intestine. GI
epithelial cells may regenerate after 4 to 6 days if there is
massive plasma replacement; antibiotics may keep patients alive
during this period. However, hematopoietic failure ensues within 2
or 3 wk and is usually fatal.
The hematopoietic syndrome is produced by whole-body doses of 2 to
10 Gy and initially causes anorexia, apathy, nausea, and vomiting.
These symptoms may be maximal within 6 to 12 h, subsiding completely
within 24 to 36 h after exposure. However, during this period of
relative well-being, lymph nodes, spleen, and bone marrow begin to
atrophy, leading to pancytopenia. Atrophy results from direct
killing of radiosensitive cells and from inhibition of new cell
production. In the peripheral blood, lymphopenia develops
immediately, becoming maximal within 24 to 36 h. Neutropenia
develops more slowly. Thrombocytopenia may be prominent within 3 or
4 wk.
In the hematopoietic syndrome, susceptibility to infection (by
saprophytic and pathogenic organisms) increases because of a
dose-dependent decrease in circulating granulocytes and lymphocytes,
dose-dependent impairment of antibody production, impairment of
granulocyte migration and phagocytosis, decreased ability of the
reticuloendothelial system to kill phagocytized bacteria, diminished
resistance to bacterial spread in subcutaneous tissues, and
development of hemorrhagic areas (due mainly to thrombocytopenia) in
the skin and bowel, which enable bacteria to enter and grow.
Acute radiation sickness: This disorder develops in a small
proportion of patients after radiation therapy (particularly to the
abdomen). Its cause is not understood. Nausea, vomiting, diarrhea,
anorexia, headache, malaise, and tachycardia of varying severity
typically occur, then subside within a few hours or days.
Intermediate delayed effects: Prolonged or repeated exposure at a
low-dose rate from internally deposited or external sources of
radiation may produce amenorrhea and decreased libido in women and
decreased fertility, anemia, leukopenia, thrombocytopenia, and
cataracts in both sexes. Higher doses or highly localized exposure
causes loss of hair, skin atrophy and ulceration, keratosis, and
telangiectasia. Ultimately, squamous cell carcinomas may develop.
Osteosarcomas may appear years after ingestion of radioactive
bone-seeking nuclides (eg, radium salts).
Extensive radiation therapy for cancer can occasionally cause
serious injury to exposed organs. If the kidneys are irradiated, GFR
and renal tubular function may decrease. Extremely high doses may
result in the acute onset of clinical manifestations (eg,
proteinuria, renal insufficiency of varying degree, anemia,
hypertension) after a latent period of 6 mo to 1 yr. When cumulative
kidney exposure is > 20 Gy in 30 Gy if treatment is not spread
over a long enough period. Extensive radiation therapy to the
mediastinum can produce radiation pericarditis and myocarditis.
Catastrophic myelopathy may develop after a cumulative dose of > 50
Gy to a segment of the spinal cord. However, this risk can be
minimized by limiting the dose rate to 2 Gy/day. If the rate is 8
Gy/day, myelopathy may occur at a cumulative dose as low as 16 Gy
(after 2 days of treatment). After extensive radiation of abdominal
lymph nodes (eg, for seminoma, lymphoma, or ovarian carcinoma),
chronic ulceration, fibrosis, and perforation of the bowel may
develop. The use of high-energy photons (which penetrate deeply into
tissues) in cobalt-60 units and linear accelerators has virtually
eliminated the skin erythema and ulceration that occurred when
orthovoltage x-ray therapy was used.
Late somatic and genetic effects: Radiation of somatic cells may
result in diseases--such as cancer (eg, leukemia; thyroid, skin, or
bone cancer) and cataracts--or, as suggested by animal models, in
nonspecific shortening of life. Thyroid cancer may occur 20 to 30 yr
after x-ray therapy for adenoid and tonsillar hypertrophy.
Externally delivered radiation appears to have a greater biologic
effect than radioiodine.
Radiation to germ cells affects the genes, and mutations increase.
Procreation perpetuates the mutations, resulting in an increased
number of genetic defectives in subsequent generations. The
long-term probability of a measurable genetic or somatic effect
appearing in a given individual is estimated to be 10-2/Gy.
Diagnosis and Prognosis
When the cerebral or GI syndrome is present, diagnosis is simple,
but the prognosis is grave. Death results within hours to a few days
in the cerebral syndrome and usually in 3 to 10 days in the GI
syndrome. In the hematopoietic syndrome, death may occur in 8 to 50
days, in 2 to 4 wk due to a supervening infection, or in 3 to 6 wk
due to massive hemorrhage. GI or hematopoietic malfunction is fatal
if the acute whole-body dose is > 6 Gy, but if the dose is 2 Gy, tissue type should
be determined, and a compatible bone marrow donor should be sought.
A marrow transplant from an identical twin increases the likelihood
of survival. If granulocytes and platelets fall to < 500/µL and <
20,000/µL, respectively, homotransplantation of marrow should be
considered, although the likelihood of success is small and
transplantation may be followed by a potentially fatal immunologic
graft-vs.-host reaction (see Bone Marrow Transplantation in Ch.
149).
Symptoms of radiation sickness due to radiation therapy of the
abdomen can be reduced by an antiemetic (eg, prochlorperazine 5 to
10 mg po or IM qid) and may be prevented by prior administration.
Ondansetron and granisetron, used for symptoms caused by
chemotherapy, may also help in radiation sickness but are much more
expensive. The radiotherapist and referring physician must cooperate
closely, giving attention to nutrition and fluid balance. Careful
planning of overall management (eg, dose, interval between
treatments, supportive therapy) can prevent most problems.
For severe chronic exposure, removing the patient from the radiation
source is the first step. If radium, thorium, or radiostrontium is
deposited in the body, prompt administration of oral and parenteral
chelating drugs (eg, EDTA) increases excretion. However, in the late
stages, these drugs are useless. Radiation ulcers and cancers
require surgical removal and plastic repair. Radiation-induced
leukemia is treated in the same way as a similar spontaneous
leukemia. Whole-blood transfusion can correct anemia, and platelet
transfusions may reduce thrombocytopenic bleeding. However, the
value of these measures is only temporary because the probability
that extensively damaged bone marrow will regenerate is slight. No
effective treatment of sterility or of ovarian and testicular
dysfunction, except for hormonal supplementation, is available.

Weapons Neutron Research Facility

Weapons Neutron Research Facility

Facility Directory Facility Description
The Los Alamos Neutron Science Center (LANSCE), which produces intense
sources of pulsed neutrons, provides the scientific community with the
capability to perform experiments supporting national security and
civilian research. The neutron beams are available at the Manuel Lujan Jr.
Neutron Scattering Center (MLNSC) – which provides neutron beams from 10-5
eV to a few hundred keV of energy – and the Weapons Neutron Research (WNR)
facility. The WNR facility consists of a high-energy “white” neutron
source (Target 4), a proton reaction area (Target-2), as well as the
nuclear physics flight paths at the MLNSC. A continuous-energy spectrum of
neutrons is produced at Target-4 via spallation reactions on a bare
unmoderated tungsten target. The energy range of the neutrons produced is
from less than 1 MeV to over 600 MeV. Because the proton beam is pulsed,
the energy of the neutrons can be determined by time-of-flight (TOF)
techniques. The time structure of the proton beam can easily be optimized
for the requirements of particular experiments. Typically, Target-4
operates with a proton beam current of approximately 5 µmA, 1.8 msec
between pulses and approximately 35,000 pulses/sec. Target-4 is the most
intense high-energy neutron source in the world. Target-4 has 6 flight
paths instrumented for a variety of measurements.
Equipment
List Not Available
Facility Access
Open
Last Update: 30 June 2003
Technical Contact:
Bruce Takala
Mail Stop: H855
Phone: 505-665-2029
Fax: 505-665-3705
E-mail Address: takala@lanl.gov User Facility Agreement Contact
Kim Sherwood
Technology Transfer Division
Mail Stop: C334
Phone: 505-665-1305
Fax: 505-665-0154
E-mail Address: ksherwood@lanl.gov


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CodeBlueBlogMD

I have a clue. It's called 2 PhD's. YOU need a clue + about 12 more years of education.

You remind me of a medical student who has a fever and goes to the Merck Manual, looks up fever and finds it under lymphoma then believes he has lymphoma.

There is no way Arafat could be exposed to the necessary dose of radiation without exposing anyone else or without the source being detected.

He certainly did not manifest acute radiation sickness so then you are saying that someone was able to selectively irradiate him over a long period of time with a low dose of radiation...by what mechanism?? maybe a UFO is involved?

Robert Stewart

A single exposure to a high power neutron beam or slightly defocused x ray laser would be sufficient to irradiate his entire body in a single dose without affecting others and would not be detected unless someone was specifically looking and leave little trace. Hitting a fixed target in bed is trivial compared to shooting as missiles, it is not unreasonable to believe the directed energy weapons folks have the capacity to do such a thing. It is also possible that someone was working on a dirty bomb or nuke and things went badly, in which case there were others exposed. I don't think the terrorists would advertise such an oops; they would just dispose of the bodies, with the exception of Arafat, his absence could not be easily explained. Either scenario is at least possible. Take a look at his symptoms one more time and tell me that they are not consistent with a single large radiation exposure.

CodeBlueBlogMD

Look....

I'm glad you are at my site reading this stuff, and you are a smart guy, way smarter than 99% of people who THINK they know what they are talking about...I appreciate your intellectual curiosity; really, and I apologize for being cynical towards you...I mean I get a LOT of crazy mail that I delete WAY befoe it gets to this site...

But let me tell you what I mean...

If you expose someone to a BIG dose of radiation (now, I am a RADIOLOGIST and I'm not sure what you mean by a defocused X ray, which is a nonsensical statement to me...)...enough to make someone SICK from a single dose...those people are sick as HELL. They vomit all the time, have constant diarrhea, and are generally INCREDIBLY miserable. I have seen these patients and attended to them on bone marrow transplant services. NO WAY Arafat had this. No way.

Hit a fixed target in bed? No Robert, no chance. An Xray beam is not a supernatural force, it is still, only, a portion of the electromagnetic spectrum and as such its behavior is relatively predictable...

Arafat may have been poisoned, but not from gamma or beta radiation...sorry.

I have taken care of these patients on a bone marrow transplant ward where we give patients acute radiation sickness on PURPOSE...and they are SO sick they can't move.

That was my point when I said ARAFAT no way had acute radiation sickness. No doctor would miss that.

Ted Christian

hey-
This concerns the Arafat/death ray thread. When you wrote:
Hit a fixed target in bed? No Robert, no chance. An Xray beam is not a supernatural force, it is still, only, a portion of the electromagnetic spectrum and as such its behavior is relatively predictable...

What's so implausible about hitting a fixed target with X or gamma radiation? It seems to me a reasonable enough proposition to produce a beam with a reasonably tight footprint and with a high enough frequency to get enough energy through intervening material and into the target. Am I missing something?
I'm no radiologist (aerospace engineer), but I generally agree that acute radiation sickness would be a no brainer diagnosis. Still, it seems to me that antipersonel directed energy weapons are on the immediate horizon if they aren't here already, and it also seems they will be much easier to fabricate than nuclear weapons. Makes me wonder how much longer we will see infantry wandering about.

CodeBlueBlogMD

What would be the source of the radiation (how would you generate the gamma radiation)? And, how would you keep the beam focused? Then, how would you aim it? And if you could do that, how would you prevent "scatter" of such an obviously powerful irradiation from affecting everyone in the vicinity?

Think of the X-ray beam as the water spray from the nozzle of a garden hose. Same problems: generation of power; maintaining focus; assuring accuracy; preventing scatter from getting everyone wet.

melissa

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